Key result
A protein implicated in the preservation of cardiomyocytes during myocardial infarction may also promote fibrosis.
Key points are not available for this paper at this time.
Warrants caution against targeting the protein therapeutically after MI; leaves open net effects on remodeling and outcomes.
A protein implicated in the preservation of cardiomyocytes during infarction may also promote fibrosis.
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Bodh I. Jugdutt (2009) studied this question. A protein implicated in the preservation of cardiomyocytes during myocardial infarction may also promote fibrosis.
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