Key result
Inflammation assessed by epicardial adipose tissue and serum biomarkers serves as an integral mechanistic link in the pathogenesis of isolated coronary artery ectasia.
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Epicardial adipose tissue hyperinflammation is proposed as a key mechanistic link in the pathogenesis of isolated coronary artery ectasia.
Inflammation, as assessed by epicardial adipose tissue and serum biomarkers, represents an integral mechanistic link in isolated coronary artery ectasia (iCAE) pathogenesis. This should be further investigated in the context of iCAE management and risk stratification.
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Papathanasiou et al. (2022) studied this question. Inflammation assessed by epicardial adipose tissue and serum biomarkers serves as an integral mechanistic link in the pathogenesis of isolated coronary artery ectasia.
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