Key result
In a patient with refractory atrioventricular nodal reentry tachycardia, reactive hypoglycemia triggered arrhythmia aggravation, including easy tachycardia initiation and failure of pacing termination.
Why the study?
Does reactive hypoglycemia trigger aggravation of arrhythmia in a patient with refractory atrioventricular nodal reentry tachycardia?
Case Report (n=1)
Does reactive hypoglycemia trigger aggravation of arrhythmia in a patient with refractory atrioventricular nodal reentry tachycardia?
This case report demonstrates that reactive hypoglycemia can trigger and aggravate arrhythmias, including AVNRT and atrial fibrillation.
Hypoglycemia evaluation may be considered in refractory AVNRT; hypothesis-generating case report that leaves open causality and generalizability.
A patient with refractory atrioventricular nodal reentry tachycardia is reported in whom it was possible to document that reactive hypoglycemia was the trigger for aggravation of arrhythmia. Over a period of 6 years, a series of electrophysiological studies revealed that, when the patient was in a hypoglycemic state, initiation of tachycardia was easy and most importantly that tachycardla termination by extra-stimulus pacing always failed. Furthermore, atrial fibrillation was inducible or spontaneously occurred only when the blood glucose level was reduced by IV insulin administration.
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Rokas et al. (1992) conducted a case report in Refractory atrioventricular nodal reentry tachycardia (n=1). Reactive hypoglycemia was evaluated on Aggravation of arrhythmia (initiation of tachycardia, failure of termination, atrial fibrillation). In a patient with refractory atrioventricular nodal reentry tachycardia, reactive hypoglycemia triggered arrhythmia aggravation, including easy tachycardia initiation and failure of pacing termination.
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