Key result
Functional neuromuscular stimulation-induced knee extension exercise elicited significant increases in oxygen uptake (130%), pulmonary ventilation (120%), and cardiac output (32%) compared to rest (p<0.05).
Why the study?
Does functional neuromuscular stimulation-induced knee extension exercise alter acute hemodynamic responses in spinal cord injured individuals?
Does functional neuromuscular stimulation-induced knee extension exercise alter acute hemodynamic responses in spinal cord injured individuals?
p-value: p=<0.05
Graded FNS-KE exercise induces small but appropriate increases in aerobic metabolism and cardiopulmonary responses in spinal cord injured individuals, though it may not be an effective central cardiovascular training tool for most.
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May elicit modest acute cardiopulmonary responses in SCI; leaves open utility as central cardiovascular training tool.
Figoni et al. (1991) studied Spinal cord injury (n=14). Functional neuromuscular stimulation-induced knee extension (FNS-KE) exercise vs. Rest was evaluated on Acute physiologic and hemodynamic responses (including oxygen uptake, pulmonary ventilation, and cardiac output) (p=<0.05). Functional neuromuscular stimulation-induced knee extension exercise elicited significant increases in oxygen uptake (130%), pulmonary ventilation (120%), and cardiac output (32%) compared to rest (p<0.05).
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