Key result
Exercise-induced syncope in a patient with severe aortic stenosis was associated with profound hypotension and sinus tachycardia, suggesting peripheral vasodilatation and ischemic myocardial depression.
Case Report (n=1)
This case report demonstrates that exercise-induced syncope in severe aortic stenosis is driven by profound hypotension likely due to peripheral vasodilatation and ischemic myocardial depression.
Answers are grounded in this paper and cite their sources.
Should not change practice in aortic stenosis; hypothesis-generating for mechanisms of exercise syncope.
A sixty-six-year-old man with severe aortic stenosis, in whom simultaneous and continuous monitoring of the electrocardiogram and arterial blood pressure was performed immediately before, during, and following recovery from exercise-induced syncope, is reported. During exercise the patient developed anginal pain associated with a gradual fall of arterial blood pressure followed by syncope. At the onset of the syncope, arterial blood pressure was extremely low, and the electrocardiogram showed sinus tachycardia. It was assumed from these findings that profound hypotension, induced by peripheral vasodilatation and ischemic myocardial depression leading to inappropriately low cardiac output, was the underlying factor mainly responsible for the occurrence of exercise-induced syncope in the present case.
No takes yet. Share an insight, caveat, or question.
Yano et al. (1989) conducted a case report in Severe aortic stenosis (n=1). Exercise was evaluated. Exercise-induced syncope in a patient with severe aortic stenosis was associated with profound hypotension and sinus tachycardia, suggesting peripheral vasodilatation and ischemic myocardial depression.
Synapse has enriched 10 related papers. Consider them for comparative context: