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May 1, 1990The Journal of Experimental MedicineOpen Access

Genetically determined lack of CD45R- T cells in healthy individuals. Evidence for a regulatory polymorphism of CD45R antigen expression.

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Authors

RSReinhard SchwinzerMedizinische Hochschule HannoverKWK WonigeitMayo Clinic

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Cite This Study

Schwinzer et al. (1990) studied this question.

synapsesocial.com/papers/6a9e56b2a5748264e86aaf99https://doi.org/10.1084/jem.171.5.1803
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Characterization of CD45 and CD45R monoclonal antibodies using transfected mouse cell lines that express individual human leukocyte common antigens.1988 · 137 citations
  2. 2Antibodies to common leukocyte antigen p220 influence human T cell proliferation by modifying IL 2 receptor expression.1985 · 164 citations
  3. 3Loss of CD45R and gain of UCHL1 reactivity is a feature of primed T cells.1988 · 928 citations
  4. 4The isolation and characterization of the human suppressor inducer T cell subset.1985 · 856 citations
  5. 5Loss of CD45R (Lp220) represents a post-thymic T cell differentiation event.1988 · 217 citations