Key result
A case report describes a female newborn with intra-uterine myocardial infarction, featuring coronary arterial lesions similar to adult coronary disease.
Case Report (n=1)
This case report documents a rare instance of intra-uterine myocardial infarction with coronary lesions resembling adult coronary artery disease.
Alerts clinicians to rare fetal MI with adult-like lesions; leaves open prenatal atherosclerosis mechanisms.
MYOCARDIAL infarction in the newborn is a rare event. Ravich and Rosenblatt reported 2 cases in 1947. 1 Subsequent reports have related such neonatal infarcts to an aberrant origin or other abnormalities of the coronary arteries such as medial calcific sclerosis, focal intimal proliferation, or subintimal fibrosis. 2-6 Most of these coronary lesions presumably develop during fetal life, and yet only one report has appeared of myocardial infarction in utero. 2 The current report records a second case of myocardial infarction in utero. The coronary arterial lesion proved of particular interest because of its similarity to lesions sometimes seen in adult coronary arterial disease. Report of a Case A 24-year-old, RH negative, gravida 2, para 1, white female was delivered of a 7 lb., 10 oz. (3,458.6 gm.) female following a pregnancy complicated only by a 36-lb. (18 kg.) weight gain. Labor was oxytocin (Pitocin) induced and lasted 4½ hours.
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James F. Clapp (1961) conducted a case report in Intra-uterine myocardial infarction (n=1). A case report describes a female newborn with intra-uterine myocardial infarction, featuring coronary arterial lesions similar to adult coronary disease.
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