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February 7, 2009AJP Heart and Circulatory Physiology

Transient opening of mitochondrial permeability transition pore by reactive oxygen species protects myocardium from ischemia-reperfusion injury

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Authors

MSMasao SaotomeHamamatsu University School of MedicineHKHideki KatohFujita Health UniversityYYYasuhiro YaguchiHamamatsu University School of Medicine

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Saotome et al. (2009) studied this question.

synapsesocial.com/papers/6a9faf671cf0e01ec7bab487https://doi.org/10.1152/ajpheart.00436.2008
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Mitochondrial Transport of Cations: Channels, Exchangers, and Permeability Transition1999 · 1,542 citations
  2. 2Mitochondrial membrane potential modulates regulation of mitochondrial Ca<sup>2+</sup>in rat ventricular myocytes2004 · 78 citations
  3. 3CaMKII-dependent reactivation of SR Ca<sup>2+</sup>uptake and contractile recovery during intracellular acidosis2002 · 38 citations
  4. 4Protective Effects of Hydrogen Peroxide Against Ischemia/Reperfusion Injury in Perfused Rat Hearts.2003 · 45 citations