Key result
Vector translating methods in 70 cases of pre-excitation syndrome demonstrated a ventricular conduction defect without QRS prolongation in about 50% of cases, favoring the bridge hypothesis.
Observational (n=70)
Vector translating methods in WPW syndrome suggest that pre-excitation is due to atrioventricular bridges rather than accelerated AV conduction.
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May support atrioventricular bridges in pre-excitation; leaves open prospective validation before clinical adoption.
Seventy cases of pre-excitation were studied by vector translating methods. Certain properties of the delta event are described that are useful in the recognition of this syndrome electrocardiographically. In about half the cases a ventricular conduction defect without QRS-interval prolongation was demonstrated to have accompanied the pre-excitation. The type of ventricular conduction defect produced depended upon the particular direction of spread of the pre-excitation wave. This and other findings are incompatible with the hypothesis that pre-excitation is due to accelerated atrioventricular conduction and favor the hypothesis that it is due to bridges, whether morphologic or electronic, between atria and ventricles.
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Grant et al. (1958) conducted an observational in Pre-excitation syndrome (Wolff-Parkinson-White) (n=70). Pre-excitation syndrome was evaluated on Ventricular conduction defect without QRS-interval prolongation. Vector translating methods in 70 cases of pre-excitation syndrome demonstrated a ventricular conduction defect without QRS prolongation in about 50% of cases, favoring the bridge hypothesis.
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