Key result
In a 4-month-old infant with H1N1-associated dilated cardiomyopathy, treatment with oseltamivir and heart failure medications improved left ventricular ejection fraction from 32% to 50% at 6 months.
Case Report (n=1)
No
H1N1 infection can rarely cause severe dilated cardiomyopathy in infants, which may be reversible with antiviral and supportive heart failure therapy.
Raises awareness for cardiac complications in H1N1-infected infants; extends sparse case literature but remains hypothesis-generating.
The symptoms and findings of influenza A (H1N1) resemble the symptoms and findings of seasonal influenza and it generally emerges as an upper respiratory tract disease. Although the majority of patients with the influenza A virus recover spontaneously without complications, there have been occasional reports of myopericarditis. However the most frequent complications have been reported as viral pneumonia and more rarely dilated cardiomyopathy. In this paper, we report a 4-month-old infant, who admitted with shortness of breath, cough, tachycardia and respiratory problems and was diagnosed as having developed dilated cardiomyopathy associated with H1N1.
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Türe et al. (2018) conducted a case report in Dilated cardiomyopathy associated with H1N1 infection (n=1). Oseltamivir and heart failure medications was evaluated on Left ventricular ejection fraction (LVEF). In a 4-month-old infant with H1N1-associated dilated cardiomyopathy, treatment with oseltamivir and heart failure medications improved left ventricular ejection fraction from 32% to 50% at 6 months.
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