There has been increasing evidence to support that COVID-19 infection is associated with hypercoagulable complications due to a systemic inflammatory response indirectly activating the coagulation cascade. This phenomenon has been termed, COVID-19-associated coagulopathy. Uniquely, trauma patients are highly susceptible to thromboembolic events. Monitoring for adequate chemical prophylaxis against these events is difficult, especially with superimposed COVID-19 infection.
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E. et al. (2023) studied this question.
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