Investigations of HL‐A genotypes and MLR reactivity were performed in two families with an increased frequency of sarcoidosis. No significant correlation between either HL‐A or MLR alleles and disease was demonstrated. Individuals possibly homozygous for MLR alleles were found in this material. A possibly impaired stimulatory capacity of cells from sarcoidosis patients is discussed.
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Möller et al. (1974) studied this question.
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