Severe Aortic Stenosis and Acute Hip Fracture

Figures

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  Figure 1. Overlapping Mechanisms Linking AS and Osteoporotic HFx Schematic illustration of the shared clinical associations and potential etiologic pathways in patients with concurrent severe aortic stenosis (AS) and hip fracture (HFx). CAD = coronary artery disease; PAD = peripheral artery disease.

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  Figure 2. Preprocedural Computed Tomography Assessment for Transcatheter Aortic Valve Replacement (A and B) Multiplanar reconstruction of the aortic annulus showing an area of 575.2 mm2 and a perimeter of 86.3 mm, guiding valve sizing. (C) Measurement of right coronary ostial height from the aortic annular plane. (D) Measurement of left coronary ostial height from the annular plane. (E) Three-dimensional volume-rendered reconstruction of the aorta and iliofemoral vasculature demonstrating transfemoral access feasibility, including vessel caliber, calcification burden, and tortuosity, with cross-sectional measurements of the iliac and femoral arteries.

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  Figure 3. Continuous Wave Doppler of the Aortic Valve Before and After TAVR Continuous wave Doppler tracings showing marked hemodynamic improvement after transcatheter aortic valve replacement (TAVR). (Left) Pre-TAVR: peak velocity 4.9 m/s, mean gradient 54 mm Hg, consistent with severe aortic stenosis. (Right) Post-TAVR: peak velocity 2.9 m/s, mean gradient 16 mm Hg, indicating a well-functioning bioprosthesis without residual stenosis or paravalvular leak.

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  Visual Summary. Severe AS and Acute HFx: An Expedited TAVR-First Pathway Overview of the multidisciplinary management strategy for concurrent severe aortic stenosis (AS) and hip fracture (HFx). TAVR = transcatheter aortic valve replacement; VTE = venous thromboembolism.