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IN 1937 Butler1 reported the removal of a pyelonephritic kidney in a patient who had hypertension and found that the hypertension improved. This was not totally unsuspected in view of the earlier work of Goldblatt et al.2 demonstrating in dogs that the kidney could produce hypertension. There remains little doubt that disease of one kidney, whether it is due to infection, impairment of blood supply or trauma, with the formation of scar tissue, may produce hypertension. As an increased number of cases of hypertension caused by unilateral renal disorders has accumulated, attention has been focused on the blood flow through . . .
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Zimmerman et al. (1961) studied this question.
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