Key result
Lp(a) may self-regulate its fibrinolysis interference by reducing platelet aggregation.
This review highlights the complex and potentially self-regulating role of Lp(a) in thrombosis, where its anti-fibrinolytic properties may be counterbalanced by its ability to reduce platelet aggregation.
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Lp(a) self-regulation of fibrinolysis may temper thrombotic risk estimates; leaves open clinical relevance pending confirmatory in vivo studies.
D. Edward Barre (2007) reported a review. Lipoprotein (a) was evaluated. Lipoprotein (a) may self-regulate its interference with fibrinolysis by reducing platelet aggregation, though conflicting in vitro and in vivo data necessitate further in vivo studies.
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