Key result
In two young patients with head injuries, neurogenic pulmonary oedema developed, highlighting the role of raised intracranial pressure and autonomic reflex activity in its pathogenesis and treatment.
Case Report (n=2)
Highlights the role of raised intracranial pressure and sympathetic autonomic reflex in neurogenic pulmonary oedema, proposing a targeted therapeutic regime.
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Supports targeted ICP reduction and sympathetic blockade in neurogenic pulmonary oedema; leaves open need for prospective validation.
Two young patients with head injuries subsequently developed neurogenic pulmonary oedema. The origin and pathways of the reflex response to cerebral trauma are discussed, with emphasis on the role of raised intracranial pressure and the sympathetic division of the autonomic nervous system in precipitating movement of systemic circulatory volume into the pulmonary circulation. Therapeutic measures are discussed which correct the progression of this condition by reducing intracranial pressure and blocking the systemic effects of autonomic reflex activity. This leads to the basis of a simple regime to treat future cases.
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Wauchob et al. (1984) conducted a case report in Head injuries and neurogenic pulmonary oedema (n=2). Therapeutic measures reducing intracranial pressure and blocking autonomic reflex activity was evaluated. In two young patients with head injuries, neurogenic pulmonary oedema developed, highlighting the role of raised intracranial pressure and autonomic reflex activity in its pathogenesis and treatment.
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