Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
March 1, 1996CellOpen Access

Wiskott–Aldrich Syndrome Protein, a Novel Effector for the GTPase CDC42Hs, Is Implicated in Actin Polymerization

View Full Paper
Ask AI
Bookmark
Share

Authors

MSMarc SymonsJDJonathan M.J. DerryBKBrian Karlak

Discussion

Loading...

Member takes

Overview

Key Points

Key points are not available for this paper at this time.

Cite This Study

Symons et al. (1996) studied this question.

synapsesocial.com/papers/6aa2d771375ea2a2e2fdd836https://doi.org/10.1016/s0092-8674(00)81050-8
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Actin polymerization and pseudopod reorganization accompany anti-CD3-induced growth arrest in Jurkat T cells.1993 · 112 citations
  2. 2Activation of phosphoinositide 3-kinase activity by Cdc42Hs binding to p85.1994 · 396 citations
  3. 3Identification and Molecular Cloning of a p21cdc42/rac1-activated Serine/Threonine Kinase That Is Rapidly Activated by Thrombin in Platelets1995 · 131 citations