Key result
Psychophysiological and urodynamic studies support the hypothesis that the sympathetic nervous system affects bladder function in prostatodynia, but lack objective proof of direct adrenergic causality.
Current evidence supports a potential role of the sympathetic nervous system in lower urinary tract dysfunction but lacks definitive proof of direct adrenergic mechanisms.
Careful clinical and investigative protocols allow the identification of a homogeneous group of patients with prostatodynia or 'anxious bladder syndrome' (underactive detrusor function). Psychophysiological studies and urodynamic measurements in such patients demonstrate remarkably consistent results, which, whilst lending support to the hypothesis that the sympathetic nervous system may effect bladder and urethral function, do not offer objective proof that adrenergic mechanisms are directly responsible for the observed dysfunctional state. observed dysfunctional state.
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GEORGE et al. (1986) conducted a review in Prostatodynia or anxious bladder syndrome (underactive detrusor function). Sympathetic nervous system / adrenergic mechanisms was evaluated. Psychophysiological and urodynamic studies support the hypothesis that the sympathetic nervous system affects bladder function in prostatodynia, but lack objective proof of direct adrenergic causality.
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