Key result
Apical scar ablation eliminates inducible VT in a patient with repaired double-chambered right ventricle.
Case Report (n=1)
Ablation of an apical scar resulting from sustained pressure overload can successfully treat recurrent ventricular tachycardia in patients with repaired double chambered right ventricle.
May support apical scar ablation for post-DCRV VT; leaves open pressure-overload scarring as a general arrhythmogenic mechanism.
A 35 year old female presented with recurrent ventricular tachycardia 5 years after she had undergone surgical repair of double chambered right ventricle. Electroanatomical mapping showed a localised scar in the apex with double potentials and good pace map. Ablation here resulted in non-inducibility of ventricular tachycardia. We hypothesise that the scarring in the apex is the result of sustained pressure overload and becomes arrhythmogenic similar to the apical scar in patients with mid-ventricular hypertrophic cardiomyopathy.
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Selvaraj et al. (2012) conducted a case report in Recurrent ventricular tachycardia after surgical repair of double chambered right ventricle (n=1). Electroanatomical mapping and ablation was evaluated on Non-inducibility of ventricular tachycardia. Electroanatomical mapping and ablation of a localized apical scar resulted in non-inducibility of ventricular tachycardia in a patient with repaired double chambered right ventricle.
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