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May 1, 1998Current BiologyOpen Access

Defects in actin-cap formation in Vav-deficient mice implicate an actin requirement for lymphocyte signal transduction

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Authors

LHLeslie J. HolsingerNorthwestern UniversityIGIsabella A. GraefStanford UniversityWSWojciech SwatSilicon Labs (United States)

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Cite This Study

Holsinger et al. (1998) studied this question.

synapsesocial.com/papers/6aa3fb6122af0bded4f43447https://doi.org/10.1016/s0960-9822(98)70225-8
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