Key result
Acute pulmonary oedema in coronary patients was associated with tachycardia, reduced stroke volume, and increased systemic blood pressure compared to the recovery state 8-13 days later.
Why the study?
What are the haemodynamic changes in coronary patients with acute pulmonary oedema between the acute state and after recovery?
Observational (n=7)
What are the haemodynamic changes in coronary patients with acute pulmonary oedema between the acute state and after recovery?
Acute pulmonary edema in coronary patients is characterized by tachycardia, reduced stroke volume, and increased systemic blood pressure with normal cardiac output, which normalize upon recovery.
May inform acute management of coronary pulmonary edema; hypothesis-generating from small case series and leaves open prospective validation.
AbstractStudies have been made of the haemodynamics in seven coronary patients with acute pulmonary oedema without shock. Five of the patients had acute myocardial infarction. The first examination was made 3–9 hours after the appearance of pulmonary oedema. During the examination evident signs of pulmonary congestion and dyspnoea could be demonstrated. The examination was made after treatment with oxygen, Cedilanide®, aminophyllin and Pethidin®. The second examination was made 8–13 days later, after all signs of pulmonary congestion had subsided. In the acute state tachycardia, reduced stroke volume and increased systematic blood pressure at a normal cardiac output were demonstrated. The difference between the two examinations was an increase of stroke volume, a reduction of the heart rate and systemic blood pressure, and an increase of the relation between stroke volume and central blood volume.
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Knutsen et al. (1968) conducted an observational in Acute pulmonary oedema in coronary patients (n=7). Acute pulmonary oedema vs. Recovery state (8-13 days later) was evaluated on Haemodynamic changes (stroke volume, heart rate, systemic blood pressure). Acute pulmonary oedema in coronary patients was associated with tachycardia, reduced stroke volume, and increased systemic blood pressure compared to the recovery state 8-13 days later.
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