Key result
Overt hyperthyroidism links to hemodynamically unstable acute PE as the only apparent trigger in three cases.
Case Report (n=3)
Overt hyperthyroidism should be considered as a potential underlying prothrombotic condition in patients presenting with unprovoked venous thromboembolism.
Hyperthyroidism may merit consideration in unprovoked PE; case reports leave open need for prospective confirmation before practice change.
Unprovoked venous thromboembolism (VTE) is defi ned as a VTE appearing in the absence of the common risk factors including cancer, surgery, hypomobility, oestrogen use or pregnancy.Around 40% of VTE patients have unprovoked VTE in real life.However, a number of additional clinical conditions may be associated to ahypercoagulable state and induce VTE, though they are less often considered in clinical practice such as the abnormalities of the thyroid function.Here we report three relevant casereports that we found in the RIETE registry in which overt hyperthyroidism is the only apparent cause of haemodynamic unstable acute pulmonary embolism (PE).
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Micco et al. (2017) conducted a case report in Unprovoked venous thromboembolism (VTE) / acute pulmonary embolism (PE) (n=3). Overt hyperthyroidism was evaluated on Haemodynamic unstable acute pulmonary embolism (PE). Overt hyperthyroidism was identified as the only apparent cause of haemodynamic unstable acute pulmonary embolism in three case reports from the RIETE registry.
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