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April 1, 2000Genes Chromosomes and CancerOpen Access

Homozygous deletions inactivateDCC, but notMADH4/DPC4/SMAD4, in a subset of pancreatic and biliary cancers

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Authors

WHWerner HilgersUniversity of BonnJSJason Jungsik SongYonsei UniversityMHMarianne Haye

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Hilgers et al. (2000) studied this question.

synapsesocial.com/papers/6aa690fa4dd50f8a0764c795https://doi.org/10.1002/(sici)1098-2264(200004)27:4<353::aid-gcc3>3.0.co;2-5
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Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1The DCC Gene: Structural Analysis and Mutations in Colorectal Carcinomas1994 · 226 citations
  2. 2The DCC gene product induces apoptosis by a mechanism requiring receptor proteolysis1998 · 427 citations
  3. 3<i>DPC4</i> , A Candidate Tumor Suppressor Gene at Human Chromosome 18q21.11996 · 2,352 citations
  4. 4Mutations in the <i>SMAD4/DPC4</i> Gene in Juvenile Polyposis1998 · 902 citations