Key result
Subarachnoid hemorrhage mimics STEMI as the cause of cardiac arrest in a comatose resuscitated patient.
Why the study?
Electrocardiogram changes, cardiac enzyme elevations, and regional wall motion abnormalities in subarachnoid hemorrhage can mimic acute coronary syndrome and acute ST-segment elevation myocardial infarction.
Case Report (n=1)
Subarachnoid hemorrhage can present with electrocardiogram changes and regional wall motion abnormalities that mimic acute ST-segment elevation myocardial infarction after out-of-hospital cardiac arrest.
Consider subarachnoid hemorrhage in post-arrest STEMI mimics; this case leaves open systematic neurologic evaluation protocols.
Electrocardiogram changes in subarachnoid hemorrhage (SAH) have been described as ST-T changes that mimic acute coronary syndrome and even acute ST-segment elevation myocardial infarction. Elevation of cardiac enzymes and abnormality of regional myocardial wall motion have been reported frequently for SAH. We report a case of an out-of-hospital cardiac arrest survivor with high suspicion of ST-segment elevation myocardial infarction based on the electrocardiogram and bedside echocardiography, who had normal coronary arteries on emergent coronary angiography. The patient was ultimately diagnosed with SAH as a cause of out-of-hospital cardiac arrest.
No takes yet. Share an insight, caveat, or question.
Park et al. (2015) conducted a case report in Out-of-hospital cardiac arrest, Subarachnoid hemorrhage (n=1). Subarachnoid hemorrhage was evaluated on Diagnosis of the cause of cardiac arrest. In a comatose resuscitated patient presenting with signs of acute myocardial infarction, the underlying cause of cardiac arrest was identified as a subarachnoid hemorrhage.