Key result
Acute myocarditis is linked to lower HRV versus postmyocardic cardiosclerosis, reflecting vegetative imbalance.
Why the study?
Does the clinical course of non-rheumatic myocarditis affect heart rate variability parameters?
Observational (n=58)
Does the clinical course of non-rheumatic myocarditis affect heart rate variability parameters?
Acute non-rheumatic myocarditis is associated with significantly decreased heart rate variability compared to postmyocardic cardiosclerosis, indicating pronounced autonomic imbalance.
Decreased HRV in acute myocarditis signals autonomic imbalance with ventricular ectopy; leaves open whether monitoring aids risk stratification.
The same mediators of the autonomic nervous system can be both triggers and modulators of internal organs activity, and heart autonomic regulation can be best estimated by studying heart rate variability (HRV). The objective of the present research is the estimation of both time and spectral HRV parameters in patients with non-rheumatic myocarditis depending on its clinical course. 58 patients (20 men and 38 women) aged 35,3 1,3 years were examined. 22 (37,9 %) patients have established acute myocarditis, and 36 (62,1 %) - postmyocardic cardiosclerosis (PMC). HRV parameters were estimated depending on the disease stage, and on presence of heart rhythm disturbances. Patients with acute myocarditis compared to those having PMC had decreased HRV parameters (SDNN, SDANN, SDNNi, pNN50, rMSSD) refl ecting vegetative imbalance, and ventricular ecthopic activity in patients with myocarditis is associated with the activation of both sympathetic and parasympathetic system, and these changed are more expressed in patients with high ventricular extrasystole gradation.
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Nagaeva et al. (2009) conducted an observational in Non-rheumatic myocarditis (n=58). Acute myocarditis vs. Postmyocardic cardiosclerosis (PMC) was evaluated on Heart rate variability (HRV) parameters (SDNN, SDANN, SDNNi, pNN50, rMSSD). Acute myocarditis was associated with decreased heart rate variability parameters compared to postmyocardic cardiosclerosis, reflecting vegetative imbalance.
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