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August 8, 2016The Journal of Experimental MedicineOpen Access

Toxoplasma gondii TgIST co-opts host chromatin repressors dampening STAT1-dependent gene regulation and IFN-γ–mediated host defenses

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Authors

GGGabrielle GayUniversité Claude Bernard Lyon 1LBLaurence BraunCentre National de la Recherche ScientifiqueMBMarie‐Pierre Brenier‐PinchartInstitut pour l'avancée des biosciences

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Gay et al. (2016) studied this question.

synapsesocial.com/papers/6aa93acbd7d677827e255f69https://doi.org/10.1084/jem.20160340
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Also Consider

Synapse has enriched 3 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Histone Deacetylase Activity Is Required to Recruit RNA Polymerase II to the Promoters of Selected Interferon-stimulated Early Response Genes2004 · 123 citations
  2. 2Toxoplasma gondii Inhibits Gamma Interferon (IFN-γ)- and IFN-β-Induced Host Cell STAT1 Transcriptional Activity by Increasing the Association of STAT1 with DNA2013 · 73 citations
  3. 3Impaired Chromatin Remodelling at STAT1-Regulated Promoters Leads to Global Unresponsiveness of Toxoplasma gondii-Infected Macrophages to IFN-γ2012 · 85 citations