What does this research mean for the field?

HER-2/neu overexpression downregulates components of the MHC class I antigen-processing machinery, resulting in an immune escape phenotype that can be reversed by IFN-gamma treatment. Novelty: ClaimNovelty.NOVEL_FINDING. Consensus alignment: ConsensusAlignment.NEUTRAL.

January 1, 2004Cancer ResearchOpen Access

HER-2/neu-Mediated Regulation of Components of the MHC Class I Antigen-Processing Pathway

View Full Paper

Ask AI

Bookmark

Authors

FHF HerrmannPaul Ehrlich Institut HLHans‐Anton LehrBoston University IDIngo DrexlerDüsseldorf University Hospital

Discussion

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Herrmann et al. (2004) studied this question.

synapsesocial.com/papers/6aa95d5fc2bf00bae84637bf https://doi.org/10.1158/0008-5472.can-2522-2

View Full Paper

Ask AI

Bookmark

January 1, 2004Cancer ResearchOpen Access

HER-2/neu-Mediated Regulation of Components of the MHC Class I Antigen-Processing Pathway

View Full Paper

Ask AI

Bookmark

Authors

FHF HerrmannPaul Ehrlich Institut HLHans‐Anton LehrBoston University IDIngo DrexlerDüsseldorf University Hospital

Discussion

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Herrmann et al. (2004) studied this question.

synapsesocial.com/papers/6aa95d5fc2bf00bae84637bf https://doi.org/10.1158/0008-5472.can-2522-2

Also Consider

Synapse has enriched 2 closely related papers on similar clinical questions. Consider them for comparative context:

View Full Paper

Ask AI

Bookmark