Key result
Review outlines embryonic cardiac lineage signaling pathways and their potential reactivation for post-injury heart repair.
Why the study?
Understanding the signaling pathways involved in embryonic heart development and their reactivation in cardiac disease may inform new strategies for cardiac repair and regeneration.
Understanding the signaling pathways involved in embryonic cardiac development and ESC differentiation may inform new strategies for cardiac repair and regeneration after injury.
Mammalian heart formation is a complex morphogenetic event that depends on the correct temporal and spatial contribution of distinct cell sources. During cardiac formation, cellular specification, differentiation, and rearrangement are tightly regulated by an intricate signaling network. Over the last years, many aspects of this network have been uncovered not only due to advances in cardiac development comprehension but also due to the use of embryonic stem cells (ESCs) in vitro model system. Additionally, several of these pathways have been shown to be functional or reactivated in the setting of cardiac disease. Knowledge withdrawn from studying heart development, ESCs differentiation, and cardiac pathophysiology may be helpful to envisage new strategies for improved cardiac repair/regeneration. In this review, we provide a comparative synopsis of the major signaling pathways required for cardiac lineage commitment in the embryo and murine ESCs. The involvement and possible reactivation of these pathways following heart injury and their role in tissue recovery will also be discussed.
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Freire et al. (2014) conducted a review in Cardiac disease and heart injury. This review summarizes major signaling pathways required for cardiac lineage commitment in embryos and embryonic stem cells, and their potential reactivation for cardiac repair following heart injury.
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