Key result
Quinidine, digitalis, and procaine amide treat arrhythmias by suppressing tachysystole or slowing ventricular rate.
This review outlines the pharmacological options for treating cardiac arrhythmias, focusing on their mechanisms of action in controlling heart rate or rhythm.
Historical antiarrhythmic strategies targeted rate or rhythm; leaves open relevance to contemporary device- and ablation-based care.
The therapy of this group of ar1hythmias centers around (1uinidine, digitalis, vagotonic agents and procaine amide.Potassium, cal.cium, fagarine, magnesium, and procaine hydrochloride have less importance but wvill also be discussed.T naures either sup- press the t chysystoli hereupon the sinus node resumes its role-as pacemaker, or they slow the ventricular rate.The deleterious h oiiidamic action of an arrhythmia is proportional to the ventricular rate.56
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David Scherf (1953) conducted a review in Cardiac Arrhythmias. Antiarrhythmic agents (quinidine, digitalis, vagotonic agents, procaine amide) was evaluated. Therapy for cardiac arrhythmias centers around quinidine, digitalis, vagotonic agents, and procaine amide, which act by suppressing tachysystole or slowing the ventricular rate.
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