Why the study?
Protein tyrosine phosphatase: phospho-protein complex structure determination remains challenging due to transient binding interactions and the need to understand specificity at the protein level.
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This integrative structural biology approach elucidates transient protein surface interactions, specifically revealing a distal encounter site for rPTPε and phospho-Src complex formation.
Protein tyrosine phosphatase: phospho-protein complex structure determination, which requires to understand how specificity is achieved at the protein level remains a significant challenge for protein crystallography and cryoEM due to the transient nature of binding interactions. Using rPTPεD1 and phospho-SrcKD as a model system, we established an integrative workflow involving protein crystallography, SAXS and pTyr-tailored MD simulations to reveal the complex formed between rPTPεD1 and phospho-SrcKD, revealing transient protein–protein interactions distal to the active site. To support our finding, we determined the associate rate between rPTPεD1 and phospho-SrcKD and showed that a single mutation on rPTPεD1 disrupts this transient interaction, resulting in the reduction of association rate and activity. Our simulations suggest that rPTPεD1 employs a binding mechanism involving conformational change prior to the engagement of cSrcKD. This integrative approach is applicable to other PTP: phospho-protein complex determination and is a general approach for elucidating transient protein surface interactions.
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Yang et al. (2021) studied this question. rPTPεD1-R220E mutation vs. Wild-type rPTPεD1 was evaluated on Association rate (kon) and phosphatase activity.
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