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The cytoplasmic buffering capacity of Thiobacillus acidophilus (along with membrane properties) is responsible for the cytoplasmic pH homeostasis in metabolically compromised cells. When a large influx of H+ occurs, the cytoplasmic buffering capacity prevents drastic changes in pH; in addition, this influx, by increasing the positive membrane potential, eventually leads to a cessation of further H+ influx.
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Zychlinsky et al. (1983) studied this question.
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