Key result
Myosin-II activity drives actin filament disassembly and redistribution to generate continuous network turnover.
Why the study?
Myosin-II is known to generate contractile forces for actin network remodeling, but its role in regulating actin turnover remains less understood due to limitations in previous in vitro models.
Demonstrates that myosin-II activity drives actin network disassembly and turnover in a reconstituted minimal actin cortex.
Hypothesis-generating for myosin-II in actin turnover; leaves open mechanisms in cytoskeletal remodeling.
Dynamic reorganization of the actomyosin cytoskeleton allows a fine-tuning of cell shape that is vital to many cellular functions. It is well established that myosin-II motors generate the forces required for remodeling the cell surface by imparting contractility to actin networks. An additional, less understood, role of myosin-II in cytoskeletal dynamics is believed to be in the regulation of actin turnover; it has been proposed that myosin activity increases actin turnover in various cellular contexts, presumably by contributing to disassembly. In vitro reconstitution of actomyosin networks has confirmed the role of myosin in actin network disassembly, but factors such as diffusional constraints and the use of stabilized filaments have thus far limited the observation of myosin-assisted actin turnover in these networks. Here, we present the reconstitution of a minimal dynamic actin cortex where actin polymerization is catalyzed on the membrane in the presence of myosin-II activity. We demonstrate that myosin activity leads to disassembly and redistribution in this simplified cortex. Consequently, a new dynamic steady state emerges in which actin filaments undergo constant turnover. Our findings suggest a multi-faceted role of myosin-II in fast remodeling of the eukaryotic actin cortex.
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Sonal et al. (2018) studied this question. Myosin-II vs. Actin network without myosin-II was evaluated on Actin network turnover and disassembly. Myosin-II activity leads to disassembly and redistribution of actin filaments in a reconstituted minimal actin cortex, generating a dynamic steady state with continuous actin turnover.
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