Key result
Ethanol withdrawal increases BP and vascular oxidative stress via angiotensin II and AT1 receptors.
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-dependent mechanisms. These findings highlight the importance of angiotensin II in ethanol withdrawal-induced increase in blood pressure and vascular oxidative damage.
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Gonzaga et al. (2018) studied this question. Ethanol withdrawal increases blood pressure and vascular oxidative stress via angiotensin II and angiotensin type 1 receptor-dependent mechanisms.
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