Key result
Surgical extirpation successfully treats STEMI caused by coronary embolism from a polycythemia-induced aortic valve mass.
Why the study?
A minority of acute myocardial infarctions occur without coronary artery disease, with diverse embolic causes including nonbacterial thrombotic endocarditis making precise diagnosis challenging.
Case Report (n=1)
No
Highlights non-bacterial thrombotic endocarditis as a rare cause of STEMI in the setting of polycythemia.
May prompt NBTE evaluation in polycythemia with STEMI; leaves open optimal management strategies.
Acute myocardial infarction is a major cause of death and disability worldwide. In general 4% to 7% of all patients diagnosed with AMI do not have coronary artery disease (CAD) at coronary angiography or autopsy. Embolisation as a mechanism of myocardial infarction is well known. Large diversity of aetiologies makes the precise diagnosis challenging. Its sources can be thrombi from left sided heart cavities, paradoxal embolism, tumors (myxoma, fibroelastoma), endocarditis (infective or nonbacterial thrombotic), valves (native, prosthetic) or heart catheterization or surgery
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Judit et al. (2020) conducted a case report in STEMI and Polycythemia (n=1). Surgical extirpation of aortic valve mass was evaluated. Surgical extirpation successfully treated a 53-year-old man who presented with an inferior STEMI caused by coronary embolism from a non-bacterial thrombotic mass on a native aortic valve due to polycythemia.
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