Synapse
⌘+K
Synapse
PulseExploreClubsResearchersJournals
Instagram
HomeClubsExplore
July 2, 2012Proceedings of the National Academy of SciencesOpen Access

RIM genes differentially contribute to organizing presynaptic release sites

View Full Paper
Ask AI
Bookmark
Share

Authors

PKPascal S. KaeserHarvard UniversityLDLunbin DengPennsylvania State UniversityMFMingming FanGuangdong University of Technology

Discussion

Loading...

Member takes

Implication

Key Points

Key points are not available for this paper at this time.

Cite This Study

Kaeser et al. (2012) studied this question.

synapsesocial.com/papers/6ab227c87eee6d3252c514f9https://doi.org/10.1073/pnas.1209318109
View Full Paper
Ask AI
Bookmark
Share

Also Consider

Synapse has enriched 4 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1RIM1α and RIM1β Are Synthesized from Distinct Promoters of the RIM1 Gene to Mediate Differential But Overlapping Synaptic Functions2008 · 104 citations
  2. 2Rab3-interacting Molecule γ Isoforms Lacking the Rab3-binding Domain Induce Long Lasting Currents but Block Neurotransmitter Vesicle Anchoring in Voltage-dependent P/Q-type Ca2+ Channels2010 · 47 citations
  3. 3A family of RIM-binding proteins regulated by alternative splicing: Implications for the genesis of synaptic active zones2002 · 264 citations
  4. 4Structural Basis for a Munc13–1 Homodimer to Munc13–1/RIM Heterodimer Switch2006 · 143 citations