Rats were deprived of riboflavin for 2 months by feeding the diets lacking in this vitamin. Radioactive iron was injected to those rats intravenously to study the half time for its disappearance from plasma (PDT1/2), plasma iron turnover, red cell iron utilization and its turnover. Similar experiments were performed in normal pair-fed rats and the results obtained were compared with riboflavin deficient animals. These studies suggest that there is a disturbance in the iron utilization and this leads to defective hematopoiesis under riboflavin deficiency.
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Jamdar et al. (1968) studied this question.
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