Key result
Intrapericardial teratoma can cause fatal tamponade and in utero demise from severe hydrops.
Why the study?
Intrapericardial teratomas can cause nonimmune hydrops and fetal demise through massive pericardial effusion and tamponade, but prenatal diagnosis and intervention strategies are not well characterized.
Case Report (n=1)
Intrapericardial teratomas can cause massive pericardial effusion and compressive tamponade, leading to hydrops and in utero demise if not diagnosed and managed prenatally.
Supports early prenatal surveillance for intrapericardial teratoma to enable decompression; leaves open optimal timing and outcomes in larger cohorts.
We report a case of intrapericardial teratoma following in utero demise at 29 weeks with nonimmune hydrops. The diagnosis was strongly suggested by ultrasound findings and confirmed by fetopathology. The mechanism whereby intrapericardial teratomas may lead to hydrops and death is massive pericardial effusion responsible for compressive tamponade. When prenatal diagnosis is performed before this stage, in utero interventions can obtain decompression, and the birth can be planned with rapid and appropriate management of the neonate.
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Pachy et al. (2007) conducted a case report in Intrapericardial teratoma with nonimmune hydrops (n=1). Intrapericardial teratoma was evaluated on In utero demise. Intrapericardial teratoma can lead to massive pericardial effusion and compressive tamponade, resulting in nonimmune hydrops and in utero demise.
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