Key result
Mild COVID-19 infection is linked to delayed onset of granulomatous myocarditis.
Why the study?
The possible temporal association between cardiac sarcoidosis or giant cell myocarditis and COVID-19 infection suggests COVID-19 could trigger granulomatous myocarditis.
Case Report (n=4)
Mild COVID-19 infection may serve as a trigger for granulomatous myocarditis, including cardiac sarcoidosis and giant cell myocarditis.
No immediate clinical implications; leaves open whether COVID-19 may trigger granulomatous myocarditis.
Patients infected with SARS-CoV-2 have varying manifestations of cardiac involvement. We report four patients presenting with symptomatic cardiac sarcoidosis (CS) or giant cell myocarditis (GCM) 1-8 months after mild COVID-19. All patients received immunosuppressive therapy and improved gradually within the following months. The possible temporal association between the CS/GCM and COVID-19 infection might suggest that COVID-19 could be a trigger for granulomatous myocarditis.
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Bollano et al. (2022) conducted a case report in Cardiac sarcoidosis and giant cell myocarditis (n=4). COVID-19 infection was evaluated on Development of cardiac sarcoidosis or giant cell myocarditis. Mild COVID-19 infection may be a trigger for granulomatous myocarditis, as seen in four patients who developed symptomatic cardiac sarcoidosis or giant cell myocarditis 1-8 months post-infection.
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