Key result
Cocaine use linked to NSTEMI with peak troponin I of 12.30 ng/mL in young male.
Why the study?
Cocaine use can cause chest pain and myocardial infarction, but detailed clinical presentations and pathophysiology in young patients are not well characterized.
Case Report (n=1)
This case report highlights the association between cocaine use and NSTEMI in young patients without significant medical history.
Answers are grounded in this paper and cite their sources.
May prompt cocaine screening in young NSTEMI patients; single case leaves open causal mechanisms and management strategies.
We present the case of a 23-year-old male with no significant past medical history who experienced acute chest pain. A diagnostic workup revealed that the patient had a non-ST-segment elevation myocardial infarction. Although the patient was not forthcoming initially with his cocaine use, he did admit it later in his emergency department course. The timing of his rise and fall of troponin is presented. The pathophysiology of cocaine-related chest pain and infarction is discussed. The patient continued to make an uneventful recovery.
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Choksey et al. (2022) conducted a case report in Non-ST Segment Elevation Myocardial Infarction (n=1). Cocaine use was evaluated on Myocardial infarction. Cocaine use in a 23-year-old male with no significant cardiac history was associated with a non-ST-segment elevation myocardial infarction and a peak troponin I level of 12.30 ng/mL.
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