To the editor: Glanzmann thromboasthenia (GT) is an autosomal recessively inherited bleeding diathesis, and mutations within the genes (ITGA2B and ITGB3) that code for subunits οφ τηe αIIbβ3 integrin have been shown to be responsible for this disorder.[1][1] The GT clinical phenotype can be
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D’Andrea et al. (2013) studied this question.
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