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Demonstrates that Chlamydia pneumoniae significantly exacerbates cytomegalovirus-induced aortic inflammatory foci in normocholesterolemic mice, highlighting pathogen cooperation.
Inflammatory foci induced by murine cytomegalovirus infection in normocholesterolemic mice were present temporarily in the aortic wall, but some of these foci developed into advanced lesions that persisted late after infection. The early foci induced by virus infection were significantly exacerbated following a single inoculation with Chlamydia pneumoniae.
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Burián et al. (2001) studied this question.
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