Key result
Acute pancreatitis mimics STEMI with ~43-fold troponin elevation, resolving with conservative management.
Why the study?
Acute pancreatitis can present with ST-segment elevation and marked troponin elevation mimicking acute coronary syndrome, creating diagnostic challenges in resource-limited settings lacking advanced cardiac imaging and coronary angiography.
Case Report (n=1)
Acute pancreatitis can manifest with profound ST-segment elevation and troponin release, creating a significant diagnostic and therapeutic dilemma in resource-limited settings where invasive coronary angiography is not available to rapidly exclude myocardial infarction.
Clinicians should consider pancreatitis in STEMI mimics; this case leaves open optimal non-invasive differentiation in resource-limited settings.
Acute pancreatitis can present with ST‐segment elevation and marked troponin elevation, mimicking acute coronary syndrome. In resource‐limited settings where coronary angiography, cardiac magnetic resonance imaging, and serial high‐sensitivity troponin testing are unavailable, distinguishing acute myocardial injury associated with pancreatitis from Type 1 myocardial infarction can be challenging.
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Aweke et al. (2026) conducted a case report in Acute pancreatitis mimicking ST-elevation myocardial infarction (n=1). Acute pancreatitis was evaluated. Acute pancreatitis in a 62-year-old man mimicked ST-elevation myocardial infarction with a 42.6-fold troponin elevation, resolving with conservative management in a resource-limited setting.
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