Key result
Fulminant myocarditis can present as refractory wide complex tachycardia mimicking VT and requiring ECPR.
Why the study?
Fulminant myocarditis can present with arrhythmias and conduction abnormalities that complicate accurate rhythm diagnosis and management in cardiogenic shock.
Case Report (n=1)
Myocardial inflammation in fulminant myocarditis can produce conduction and depolarization abnormalities on EKG that mimic malignant ventricular arrhythmias.
Wide complex tachycardia in fulminant myocarditis may reflect conduction abnormalities rather than VT; leaves open need for refined EKG criteria in inflammatory states.
Fulminant myocarditis may present with malignant ventricular arrhythmias and cardiogenic shock. However, myocardial inflammation can also result in conduction and depolarization abnormalities in sinus or junctional rhythm that may interfere with an accurate rhythm diagnosis on electrocardiography (EKG). We report a case of shock-refractory wide complex tachycardia initially treated as ventricular tachycardia that ultimately represented inflammatory myocardial injury requiring extracorporeal cardiopulmonary resuscitation (E-CPR) and urgent transplant.
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Kazzi et al. (2026) conducted a case report in Fulminant myocarditis (n=1). Extracorporeal cardiopulmonary resuscitation (E-CPR) and urgent transplant was evaluated. Fulminant myocarditis presented as shock-refractory wide complex tachycardia mimicking ventricular tachycardia, ultimately requiring extracorporeal cardiopulmonary resuscitation and urgent transplant.
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