Key result
Stanford type A aortic dissection with concurrent splenic artery rupture causes rapid deterioration refractory to CPR.
Why the study?
Stanford type A acute aortic dissection complicated by concurrent splenic artery rupture is rare and challenging to treat, because aortic replacement surgery requiring extracorporeal circulation after heparinization could exacerbate the active bleeding.
Case Report (n=1)
No
Answers are grounded in this paper and cite their sources.
Highlights the extreme therapeutic dilemma of managing Stanford type A acute aortic dissection requiring heparinized cardiopulmonary bypass when complicated by concurrent active bleeding from a splenic artery rupture.
A 50-year-old man presenting with sudden-onset lumbago, right lower-extremity numbness, and vomiting was transferred to our hospital. On arrival, his right lower extremity was pale, and the right femoral artery was not palpable. Contrast-enhanced computed tomography demonstrated Stanford type A acute aortic dissection with a communicating false lumen causing obstruction of the right coronary artery ostium, superior mesenteric artery, and right iliac artery. Concurrently, an abdominal hematoma with contrast extravasation from the splenic artery was identified, indicating splenic artery rupture. Shortly after completion of the imaging examination, the patient’s condition rapidly deteriorated, and he was unresponsive to cardiopulmonary resuscitation. This case is rare and challenging to treat, considering the complicated type A acute aortic dissection and active bleeding from the splenic artery. The patient would have required aortic replacement surgery using extracorporeal circulation after heparinization, which could exacerbate the bleeding. This case emphasizes the need for treatment strategy discussion.
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Kenshi Yoshimura (2026) conducted a case report in Stanford type A acute aortic dissection with splenic artery rupture (n=1). Cardiopulmonary resuscitation was evaluated on Response to cardiopulmonary resuscitation / survival. In a 50-year-old man (n=1), Stanford type A acute aortic dissection with concurrent splenic artery rupture caused rapid deterioration unresponsive to cardiopulmonary resuscitation.
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