Key points are not available for this paper at this time.
在巨噬细胞中条件性缺失。我们的发现识别了损伤部位巨噬细胞中MERTK的切割,作为PNI后促进痛觉和组织损伤的神经免疫信号的重要调节因子。
Pandey等(2026)研究了这个问题。
Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context: