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August 4, 2025

Supplementary Figure S2 from METTL3 Promotes Gastric Cancer Progression via Modulation of FNTA-Mediated KRAS/ERK Signaling Activation

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Authors

FHFangqi HuSZSong ZhangJCJie Chai

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Overview

Quantification analysis shows mettl3 modulates fnta to activate kras/erk signaling in gastric cancer, implying a role in disease progression.

Key Points

  • MAIN FINDING: METTL3 promotes gastric cancer progression through modulation of signaling pathways.
  • KEY EVIDENCE: The study found that METTL3 affects FNTA, resulting in enhanced KRAS/ERK signaling activation.
  • APPROACH: Quantification analysis was used to assess the modulation of signaling components related to gastric cancer.
  • SIGNIFICANCE: Understanding METTL3's role in cancer can contribute to better-targeted therapies for gastric cancer.

Cite This Study

Hu et al. (2025) studied this question.

synapsesocial.com/papers/689a0f86e6551bb0af8d0988https://doi.org/10.1158/1541-7786.29819560
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Supplementary Figure S3 from METTL3 Promotes Gastric Cancer Progression via Modulation of FNTA-Mediated KRAS/ERK Signaling Activation2025
  2. 2Supplementary Figure S1 from METTL3 Promotes Gastric Cancer Progression via Modulation of FNTA-Mediated KRAS/ERK Signaling Activation2025
  3. 3Data from METTL3 Promotes Gastric Cancer Progression via Modulation of FNTA-Mediated KRAS/ERK Signaling Activation2025
  4. 4Supplementary Figure S9 from TERC Stimulates Fatty Acid Metabolism to Promote Bladder Cancer Progression2025
  5. 5Supplementary Figure S4 from TERC Stimulates Fatty Acid Metabolism to Promote Bladder Cancer Progression2025