Figure S5 from PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer

Key Points

EMT induction occurs following treatment with etomoxir and MCM, enhancing the metastatic potential.
Bulk transcriptomics highlights the role of metabolic changes in promoting tumor metastasis in pancreatic cancer.
Research showcases PPARδ's orchestrating effects on promoting a metabolic response linked to cancer progression.
Findings may guide therapeutic strategies targeting metabolic pathways in pancreatic cancer management.

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Bulk transcriptomics reveals that etomoxir and MCM induce EMT in pancreatic cancer, suggesting metabolic influence on tumor progression.

EMT induction by etomoxir and MCM in bulk transcriptomics

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Parejo-Alonso et al. (2025) studied this question.

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