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September 10, 2025Biomarkers

Oxidative DNA damage may promote the development of endometriosis by activating telomerase and extending telomere length: a meta-analysis

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Meta-analysis shows elevated oxidative dna damage markers in endometriosis patients, indicating telomerase activation's impact on telomere length.

Elevated levels of oxidative dna damage biomarkers were found in women with endometriosis, suggesting a link to disease development.
Meta-analysis of 42 studies indicated significantly increased 8-ohdg and telomerase activity in endometriosis patients compared to controls.
Higher risks of endometriosis correlated with specific genetic polymorphisms in hogg1 and xrcc4, emphasizing their roles as potential biomarkers.
Longer telomere length was identified as a significant risk factor for developing endometriosis, highlighting implications for diagnosis and treatment.

He et al. (2025) studied this question.

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Meta-analysis shows elevated oxidative dna damage markers in endometriosis patients, indicating telomerase activation's impact on telomere length.