This research reveals TQ-3959 as a potent BTK PROTAC degrader, indicating it may provide a new strategy for lymphoma treatment.
BTK is a critical nonreceptor tyrosine kinase involved in BCR signaling and represents a validated target for treating B-cell malignancies and autoimmune diseases. Although several BTK inhibitors have been approved, their clinical application is limited by drug resistance and off-target effects. PROTACs offer a promising alternative strategy by degrading BTK through the ubiquitin-proteasome system. Herein, we report the design and optimization of a new class of BTK PROTACs incorporating a novel benzisoxazole-based CRBN ligand. Compound 17 (TQ-3959) exhibited exceptional degradation potency (DC50 = 0.4 nM) and oral bioavailability (F = 58.0%) in mice. Importantly, TQ-3959 exhibited potent antiproliferative activity in vitro against multiple lymphoma cell lines and effectively inhibited tumor growth in vivo, achieving nearly complete regression in a TMD-8 xenograft mouse model. Our data demonstrate that TQ-3959 is a promising BTK PROTAC degrader for extensive evaluation as a new therapy for the treatment of lymphoma.
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Ren et al. (2025) studied this question.
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