September 28, 2025

Pro-197-his/ser mutation and the metabolic gene DsUGT84A1, synergistically confer resistance to tribenuron-methyl in Descurainia sophia.

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Descurainia sophia displays a 35.20-fold higher resistance index to tribenuron-methyl, indicating substantial adaptation to herbicide pressure.
The mutation from proline to histidine or serine at position 197 of the ALS gene contributes to target-site resistance mechanisms.
Metabolic processes involving P450 genes and the UGT gene DsUGT84A1 are elevated in resistant populations, suggesting a synergistic role in herbicide resistance.
Upregulation of antioxidant-related genes in transgenic plants highlights potential mechanisms for reducing toxicity from reactive oxygen species.

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Implication

The investigation shows elevated resistance levels in Descurainia sophia, suggesting metabolic gene upregulation contributes significantly to resistance mechanisms.

Cite This Study

Tian et al. (2025) studied this question.

synapsesocial.com/papers/68d913ab4ddcf71ba560bd56 https://doi.org/10.1111/tpj.70487

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Implication

The investigation shows elevated resistance levels in Descurainia sophia, suggesting metabolic gene upregulation contributes significantly to resistance mechanisms.

Pro-197-his/ser mutation and the metabolic gene DsUGT84A1, synergistically confer resistance to tribenuron-methyl in Descurainia sophia.

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Discussion

Implication

Cite This Study

Pro-197-his/ser mutation and the metabolic gene DsUGT84A1, synergistically confer resistance to tribenuron-methyl in Descurainia sophia.

Key Points

Discussion

Implication

Cite This Study

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